Upcoming Webinars

Biofilm is present in chronic wounds anywhere from 60% to 100% of the time. Biofilm has multiple colony defenses that must be overcome to manage chronic wounds effectively. This webinar will discuss the role of treatment modalities in the effective management of biofilm, as well as clinical presentation of wounds in discontinuing intervention as control of the wound (less exudate, less slough, etc.) is obtained.

In this Product Navigator, Dr. Michael Desvigne explains the benefits he sees when using Vashe Wound Solution (pure hypochlorous acid) as a standard treatment for non-intact skin in the hospital.

Negative Pressure Wound Therapy (NPWT) is a widely accepted modality in the treatment of complex wounds.

Explore the wound healing process with a look into the benefits added by placental tissue.

According to statistical data, more than 2.5 million patients will be impacted by pressure injuries in the United States alone per year, with estimated care for these wounds averaging around $11 mi

Hospital-acquired pressure injuries (HAPIs) continue to rise despite progress made in reducing all other hospital-acquired conditions (HACs).

Despite new wound care products and technologies, the burden of wounds for both patients and their providers continues to rise. In this environment, wound biofilm continues to emerge as a key factor in the pathology of non-healing wounds of all etiologies, and effective biofilm management as the missing link in these hard-to-heal wounds.

Preventing pressure injuries has always been a challenge, not just for the caregivers, but also for the health care system as a whole.

The cost of caring for non-healing, chronic wounds in the United States has been estimated at a staggering $50 billion per year, 10 times more than the budget of the World Health Organization.

Surgical intervention remains one of the key pillars of treatment in health care.

High bacterial loads and biofilm impede wound healing and must be removed but are challenging to detect at the point-of-care.

Transform your approach to complex wound care by participating in this program highlighting the accessibility and new science associated with negative pressure would therapy (NPWT).

With a growing, aging population with multiple health issues, chronic wounds are becoming more common, but costs are high and escalating quickly. Health care providers are asked to do more with less. Pressure is building in an already overloaded health care system.

Lower extremity ulcers, especially diabetic foot ulcers (DFUs) and venous leg ulcers (VLUs), can be difficult to manage, and wound closure sometimes takes many months to achieve.

Following admission to a facility, medical device–related pressure injuries (MDRPIs) occur more quickly than other types of pressure injuries.

Choice is good, but making the right choice is better.

We all have flora on our skin, but when the skin microbiome is out of balance because of excessive heat and moisture, serious skin problems can result.

This activity provides 1 CME credit / 1 contact hour.
Current evidence links the pathology of wound hard-to-heal wounds directly to commonplace wound biofilms. The normal healing process of wounds may become derailed due to unseen barriers such as pathogenic biofilm. Biofilms are tenacious and house a colony of multispecies microbes, however not detected by the naked eye. Patients are at higher risk of infection, impaired quality of life, and financial burden when delayed healing due to biofilm presence occurs.

Critically ill patients in intensive care units are the most disadvantaged when it comes to maintaining intact skin, starting from day one of their stay. These patients are highly vulnerable to pressure injuries because of limited mobility, the presence of multiple medical devices, and the severity of their disease, including patients with acute respiratory distress syndrome (ARDS).

Wound care clinicians understand the importance of wound cleansing; wounds are warm and moist, creating the perfect breeding ground for bacteria and biofilm.